JOURNAL OF THROMBOSIS AND THROMBOLYSIS, vol.24, no.2, pp.145-151, 2007 (SCI-Expanded)
Background Impairment of the release of endothelium and platelet derived-nitric oxide (NO) increases thrombus formation that is rich in platelets in non-ST elevation acute coronary syndromes (NSTE-ACS). Since intracoronary thrombus formation and distal embolization increases risk of myocardial injury, we studied the relationship between NO levels and Tn-I in patients with NSTE-ACS.